If you reside in an apartment or house or just would like to ensure your home is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the easiest is to restrict your use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from reaching your house. http://note.pearltrees.com/14006013/19ce1ae5877820750087b318f653d9df to shield your home against EMF radiation would be to install an RF shielding canopy. This is a fabric of net that contains EMF shielding and is used to prevent EMFs from entering a room. emf blocker is to have your home equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
Numerous studies have demonstrated how the non-ionizing energy of RF has antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed result from the deregulation in cancer-related stem cells. This could be the reason for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism behind AM RF EMF's effect in patients suffering from cancer isn't clear.
The effects on the effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumours were divided in three different groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to the one used by humans. Third group members were exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumours was evaluated against that of RCF. The results revealed that cancers treated with HCCMF had significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is mediated by CACNA1H which is a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have wider implications in the treatment and diagnosis of many cancers.
The tumors in the control group were not exposed to EMF from RF, and fed a normal diet of mice. The tumors in HCCMF HCCMF group were treated with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed in cases of excessive burden.
The tumors of the three groups also showed distinct growth curves. emf blocker for home treated with HCCMF showed a significant decrease in tumour size after eight weeks. However, tumors treated with RCF didn't show shrinkage. The difference was significant. The tumours treated with RCF showed necrosis, which is common in tumours exposed to RCF. The possibility is that the necrosis is caused by an absence of oxygen in the larger tumors.
In sum, the results indicate the fact that AM EMF has anticancer activity in vitro and in live. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF produces these effects due to CACNA1H, a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the development of HCC tumors in the vivo.